Ventricular Fibrillation and Sudden Coronary Death
The publication embodied here represents the life work of a premier Russian scientist studying Sudden Cardiac Death. As one can gather from more than 35 first authored publications cited in the References, Dr. Rajskina has been involved with the investigation of mechanisms responsible for Sudden Car...
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| Format: | eBook |
| Language: | English |
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New York, NY
Springer US
1999, 1999
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| Edition: | 1st ed. 1999 |
| Series: | Developments in Cardiovascular Medicine
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| Subjects: | |
| Online Access: | |
| Collection: | Springer Book Archives -2004 - Collection details see MPG.ReNa |
Table of Contents:
- 5.6 Comparison of the effects of coronary artery occlusion andadrenaline on metabolism and electrophysiological processes in the heart
- 6. The Prevention of Ventricular Fibrillation after Coronary Artery Occlusion
- 6.1 The main directions of the antifibrillatory influences
- 6.2 Method of computer control of the use of antifibrillatory substances
- 6.3 The methods of prognostication of ventricular fibrillation
- 6.4 Evaluation of effectiveness of antifibrillatory influences
- 6.5 Conclusions
- 7. Mechanism of Ventricular Fibrillation Onset After Coronary Artery Occlusion
- 7.1 Electrophysiological hypotheses of ventricular fibrillation mechanism
- 7.2 Metabolic hypotheses of ventricular fibrillation
- 7.3 Neurohumoral hypotheses of ventricular fibrillation
- 7.4 Author’s hypothesis based on the systemic approach to study of ventricular fibrillation mechanisms
- 7.5 Ventricular fibrillation as a result of adrenal overcompensation
- Implications
- References
- 1. The Ventricular Fibrillation and Heart’s Blood Supply
- 1.1 The types of cardiac blood supply
- 1.2 Hemodynamic changes
- 1.3 Collateral blood supply of the heart
- 1.4 The size of the ischemie zone
- 1.5 Po2 in the ischemie zone
- 1.6 Conclusion
- 2. Ventricular Fibrillation and Electrophysiological Changes
- 2.1 Role of the VPB in the onset of VF after CAO
- 2.2 Role of ionic current, membrane potentials, automatism, excitability, and conductivity changes in the appearance of VF after CAO
- 2.3 Study of mechanisms of ventricular arrhythmia using mathematical modeling and computer simulation
- 2.4 Conclusions
- 3. Ventricular Fibrillation and Changes in Metabolism of the Heart after Local Ischemia
- 3.1 Ischemic changes in the ox-red equilibrium
- 3.2 Ischemic disturbances in acid-base equilibrium
- 3.3 Ischemic disturbances in ion equilibrium
- 3.4 Conclusion
- 4. Role of the Sympathoadrenal System in the Appearance of Ventricular Fibrillation after Coronary Artery Occlusion
- 4.1 Afferent cardiac nerve activity and ventricular fibrillation after coronary artery occlusion
- 4.2 Afferent activity of the aortal nerve after CAO
- 4.3 Catecholamine balance in the heart and ventricular fibrillation after coronary artery occlusion
- 4.4 Conclusions
- 5. Role of Adrenaline in the Mechanisms of Ventricular Fibrillation Onset after Occlusion of Coronary Artery
- 5.1 Effect of adrenaline on the ox-red equilibrium of the heart
- 5.2 Effect of adrenaline on the acid-base equilibrium
- 5.3 Effect of adrenaline on ionic equilibrium
- 5.4 Influence of adrenaline on the heart’s electrophysiological processes
- 5.5 The comparison of the effects of arrhythmic and non- arrhythmic doses of adrenaline on a metabolism