Alzheimer's Disease: Cellular and Molecular Aspects of Amyloid beta
This book contains a survey of present-day research into the biomedical fundamentals of Alzheimer’s disease (AD). It contains 20 chapters dealing with widely ranging topics, all of which have a bearing upon the understanding and treatment of AD. Starting with a broad survey of the contribution that...
| Other Authors: | , |
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| Format: | eBook |
| Language: | English |
| Published: |
New York, NY
Springer US
2005, 2005
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| Edition: | 1st ed. 2005 |
| Series: | Subcellular Biochemistry
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| Subjects: | |
| Online Access: | |
| Collection: | Springer eBooks 2005- - Collection details see MPG.ReNa |
Table of Contents:
- The Contribution of Microscopy to the Study of Alzheimer’s Disease, Amyloid Plaques and A? Fibrillogenesis
- Transgenic Mouse Models for APP Processing and Alzheimer’s Disease: Early and Late Defects
- Oxidative stress in Alzheimer’s Disease: Implications for Prevention and Therapy
- ?-Secretase, APP and A? in Alzheimer’s Disease
- The Non-Amyloidogenic Pathway: Structure and Function of ?-Secretases
- Amyloid ? Degradation: A Challenging Task for Brain Peptidases
- The Protective Role of Vitamin E in Vascular Amyloid ?-Mediated Damage
- Amyloid Accumulation and Pathogensis of Alzheimer’s Disease: Significance of Monomeric, Oligomeric and Fibrillar A?
- Cholesterol and Amyloid ? Fibrillogenesis
- Alzheimer’s ?-Amyloid: Insights into Fibril Formation and Structure from Congo Red Binding
- The Aluminium-Amyloid Cascade Hypothesis and Alzheimer’s Disease
- Amyloid-? Metal Interaction and Metal Chelation
- The Interaction of Amyloid-? with ApoE
- Clusterin and Alzheimer’s Disease
- Acetylcholinesterase Interaction with Alzheimer Amyloid ?
- Membrane Disordering Effects of ?-Amyloid Peptides
- The Role of Alzheimer A? Peptides in Ion Transport Across Cell Membranes
- Amyloid Inhibitors and ?-Sheet Breakers
- Cholesterol and Alzheimer’s Disease: Statins, Cholesterol Depletion in APP Processing and A? Generation
- Phosphorylated Amyloid-?: the Toxic Intermediate in Alzheimer’s Disease Neurodegeneration